嘉義再傳毒菜脯 苯甲酸超標3倍

2013/06/12

端午節習俗還要吃粽子，不過不少粽子裡面會加的菜脯，竟然有問題，嘉義市一間40年老字號菜脯工廠，被查出添加過量的防腐劑苯甲酸，還超標三倍！這間工廠，出貨遍布全台灣，還外銷，不過日本檢驗嚴格，業者不敢加防腐劑，反而把防腐菜脯，賣給台灣人，目前工廠裡80公噸菜脯、通通被查扣。
　 　今天是端午節，也是吃粽子的節日，不過，包在粽子裡頭的菜脯，嘉義縣調站和衛生局，又查到了嘉義市區一家菜脯工廠，存放的80公噸，不但添加了防腐劑 「苯甲酸」，還超標三倍。製造菜脯有40年經驗的林姓業者說，都是靠經驗添加，所以難免會過量，只不過，外銷到日本的菜脯，因為檢驗嚴格怕被退貨，所以一 滴都不加。
　　咬一口粽子熱氣香氣全冒出來，尤其包在粽子裡的這些菜脯，脆脆的咬感是不少人的最愛，只不過又有業者被查到，在菜脯裡添加了過量的苯甲酸，就是位於嘉義市區的這間菜脯工廠，調查人員會同衛生局稽核員，來到工廠稽查裡頭堆放著，一箱箱準備出貨的菜脯，檢調人員開箱 打開一包，聞到一股刺鼻的味道。
　　因為這些菜脯裡都添加了，防腐劑也就是苯甲酸，對於添加苯甲酸，業者說是為了延長保存期限，不過添加的劑量卻被驗出，超標了三倍，這間名叫"雄"字號，製造菜脯已經有40年的老字號工廠，這次被查獲的苯甲酸菜脯，就有80公噸全被封存，只不過調查人員發現，業者出口給日本的菜脯，這苯甲酸卻是一滴都不敢加，就因為怕退貨，這真的要怪主管單位把關不夠嚴格，讓業者把安全菜脯出口，台灣人吃的卻是問題菜脯。

http://news.cts.com.tw/cts/society/201306/201306121259016.html

菜农之子 - - “亲眼看见他大瓶小瓶地买剧毒的药，怎么吃得下”

　　对于这些禁药的毒性，菜农们心知肚明，但似乎已经相当淡定。与张春新挨着菜地的王孟祥告诉新快报记者，这些自己种的菜大部分卖掉，自己也吃，“没听说 有谁吃了有问题的，就是韭菜吃了容易拉肚子”，他说，“哪里的都一样，都是有农药的，难道就不吃菜了吗？那也太讲究了吧。”

　　张春新的儿子张帆在医院工作，多年来坚持不吃父亲种的菜。“亲眼看见他大瓶小瓶地买那些剧毒的药，怎么吃得下？”张帆告诉记者，他吃的菜都是自己再去市场上买，虽然也不能保证其他人的菜是不是也有农药，但是“起码不知道实情，心里舒服些”。

　　张春新认为儿子的行为很滑稽。“谁家的不是放了药的，我放的反而是量比较少的，你去买的那些，药更多。”

　　张春新种的蔬菜除了少部分自己吃，绝大部分批量卖给市场以及本地超市。“市场上买了我的菜，装了车就运到外地了；超市里记下我的地址和电话，也就把我的菜收下了”，张春新说，除此之外，卖菜的过程没有其他的检查环节。

http://www.wenxuecity.com/news/2013/05/28/2422034.html

New Study Links GMO Food To Leukemia: GMO BT Cry-Toxins May Contribute to Blood Abnormalities

May 16, 2013     By Sayer Ji

A groundbreaking new study published in the current issue of the Journal of Hematology & Thromboembolic Diseases reveals the potential "leukemogenic" properties of the Bt toxin biopesticides engineered into the vast majority of GMO food crops already within the US food supply. 

a new study published in the Journal of Hematology& Thromboembolic Diseases indicates that the biopesticides engineered into GM crops known as Bacillus Thuringensis (Bt) or Cry-toxins, may also contribute to blood abnormalitiesfrom anemia to hematological malignancies(blood cancers) such as leukemia.
A group of scientists from the Department of Genetics and Morphology, Institute of Biological Sciences, University of Brasilia, Brasilia/DF, Brazil set out to test the purported human and environmental biosafety of GM crops, looking particularly at the role that the Bt toxin found within virtually all GM food crops plays on non-target or non-insect animal species. 

The research was spurred by the Brazilian Collegiate Board of Directors of the National Sanitary Surveillance Agency (ANVISA), who advocated in 2005 for evaluations of toxicity and pathogenicity of microbiological control agents such as Bt toxins, given that little is known about their toxicological potential in non-target organisms, including humans.

While Bacillus Thurigensis spore-crystals have been used since the late 1960's in agriculture as a foliar insecticide, it was only after the advent of recombinant DNA biotechnology that these toxin-producing genes (known as delta endotoxins) were first inserted into the plants themselves and released into commercial production in the mid-90's, making their presence in the US food supply and the bodies of exposed populations ubiquitous. 

What the new study revealed is that various binary combinations and doses of Bt toxins are capable of targeting mammalian cells, particularly the erythroid (red blood cell) lineage, resulting in red blood cell changes indicative of significant damage, such as anemia. In addition, the study found that Bt toxins suppressed bone marrow proliferation creating abnormal lymphocyte patterns consistent with some types of leukemia.

The researchers also found that one of the prevailing myths about the selective toxicity of Bt to insects, the target species, no longer holds true:

It has been reported that Cry toxins exert their toxicity when activated at alkaline pH of the digestive tract of susceptible larvae, and, because the physiology of the mammalian digestive system does not allow their activation, and no known specific receptors in mammalian  intestinal cells have been reported, the toxicity these MCAs to mammals  would negligible. However, our study demonstrated that Bt spore-crystals genetically modified to express individually Cry1Aa, Cry1Ab, Cry1Ac or Cry2A induced hematotoxicity, particularly to the erythroid lineage. This finding corroborates literature that demonstrated that alkali-solubilized Bt spore-crystals caused in vitro hemolysis in cell lines of rat, mouse, sheep, horse, and human erythrocytes and suggested that the plasma membrane of susceptible cells (erythrocytes, in this case) may be the primary target forthese toxins

The study also found:

1) That Cry toxins are capable of exerting their adverse effects when suspended in distilled water, not requiring alkalinizationvia insect physiology to become activated as formerly believed.

2) That a dose of Cry1Ab as low as 27 mg/kg, their lowest tested dose, was capable of inducing hypochromic anemia in mice – the very toxin has been detected in blood of non-pregnant women, pregnant women and their fetuses in Canada, supposedly exposed through diet.

3) Whereas past reports have found that Bt toxins are generally nontoxic and do not bioaccumulate in fatty tissue or persist in the environment, the new study demonstrated that all Cry toxins tested had a more pronounced effect from 72 hours of exposure onwards, indicating the opposite is true.

4) That high-dose Cry toxin doses caused blood changes indicative of bone marrow damage (damage to "hematopoietic stem cell or bone marrow stroma").
The authors noted their results "demonstrate leukemogenic activity for other spore-crystals not yet reported in the literature."

They concluded:

Results showed that the Bt spore-crystals genetically modified to express individually Cry1Aa, Cry1Ab, Cry1Ac or Cry2A can cause some hematological血液学risks to vertebrates脊椎动物,increasing their toxic effects with long-term exposure. Taking into account the increased risk of human and animal exposures to significant levels of these toxins, especially through diet, our results suggest that further studies are required to clarify the mechanism involved in the hematotoxicity found in mice, and to establish the toxicological risks to non-target organisms, especially mammals, before concluding that these microbiological control agents are safe for mammals.